Aldosterone, Vol 109

  • ID: 4612908
  • Book
  • 300 Pages
  • Elsevier Science and Technology
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Aldosterone, Volume 109, the latest release in the Vitamins and Hormones series first published in 1943, covers the field of hormone action, vitamin action, X-ray crystal structure, physiology and enzyme mechanisms, with this release focusing on topics relating to Aldosterone Research, Aldosterone and Micrornas, the Evolution of the Mineralocorticoid Receptor, Aldosterone and Kidney Micrornas, Adipocyte Mineralocorticoid Receptor, Regulation of Aldosterone Secretion, Leptin and Aldosterone, Cell- and Ligand-Specific Interactions in Mineralocorticoid Receptor Signaling, Primary Aldosteronism, Present and Future, Aldosterone-Producing Adenomas, Overexpression of the Mineralocorticoid Receptor, Aldosterone and Myocardial Pathology, and much more.

  • Focuses on the newest aspects of hormone action in connection with diseases, with this update focusing on aldosterone
  • Lays the groundwork for the focus of new chemotherapeutic targets
  • Represents reviews on emerging areas in hormone action, cellular regulators and signaling pathways
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Preface Gerald Litwack 1. Overview of Aldosterone Research John W. Funder 2. Aldosterone and Micrornas Diego Alvarez De La Rosa 3. Evolution of the Mineralocorticoid Receptor Michael Edward Baker 4. Aldosterone and Kidney Micrornas Michael Bruce 5. Adipocyte Mineralocorticoid Receptor Massimiliano Caprio 6. Regulation of Aldosterone Secretion Eleanor Davies 7. Leptin and Aldosterone Eric J. Belin De Chantemele 8. Cell- and Ligand-Specific Interactions in Mineralocorticoid Receptor Signalling Peter Fuller 9. Primary Aldosteronism, Present and Future John W. Funder 10. Aldosterone-Producing Adenomas Per Hellman 11. Overexpression of the Mineralocorticoid Receptor Sofia Kanatsou 12. Aldosterone and Myocardial Pathology Walter Koch 13. Paracrine Regulation of Aldosterone Secretion in Physiological and Pathophysiological Conditions Herve Lefebvre 14. Mineralocorticoid Antagonists Anneli Nordqvist
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